Cellular Aspects of Hypertension.
Cellular Aspects of Hypertension. eBook 71,68 €. price for Finland (gross). This choice of approach is due to some extent to the build up of know-how that molecular and cellular biology have been producing at a continuous rate. As the contents list of this volume shows, a large mass of work has been directed to gaining some insight into pathogenetic mechanisms.
Cellular Aspects of Hypertension book.
Cellular Mechanisms of Spontaneous Hypertension and Stroke: Role of Vascular Smooth Muscle Cells. Biomechanical and Electrical Responses of Normal and Hypertensive Veins to Short-Term Pressure Increases. Hypertrophic Growth of Vascular Smooth Muscle. II: Calcium and Circulatory Control. Dysfunction of Ca2+-pump of Vascular Muscle Membranes: An Important Etiological Factor in Hypertension. Changes in Ca2+ and Ca2+ Sensitivity During on of Arterial Muscle
Bruschi G1, Regolisti G, Borghetti A. Author information. 1. Istituto di Clinica Medica e Nefrologia, Università degli Studi di Parma. Cell calcium control alterations have been reported in different forms of hypertension.
Bruschi G1, Regolisti G, Borghetti A. The control of vasomotion is a central issue in blood pressure regulation and is a primary goal of antihypertensive therapy. An increase in vascular myoplasmic Ca++ and a higher rate of calcium influx through specific, itive calcium channels have been found in genetic or secondary animal models. In hypertensive patients, an elevation of cytoplasmic Ca++ was noted in the platelets.
The control of vasomotion is a central issue in blood pressure regulation and is a primary goal of antihypertensive therapy. Calcium is the final messenger in the contractile mechanism of vascular smooth and cardiac muscle. Cellular Aspects of Hypertension.
Автор: Giacomo Bruschi; Alberico Borghetti Название: Cellular Aspects of Hypertension ISBN: 3662009854 .
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A (eds): Cellular Aspects of Hypertension. G Bruschi, Borghetti. SpringerVerlag, Heidelberg 51-57
A (eds): Cellular Aspects of Hypertension. SpringerVerlag, Heidelberg 51-57, 1991. Monoclonal antibody Ki67 as a proliferation marker. Andreas Neubauer, Dieter Huhn.
Hypertension is common in patients with kidney disease for a number of reasons, including retention, inappropriate action by the renin-angiotensin system, and overactivity of the sympathetic nervous system. Of all causes of hypertension, renal artery stenosis (narrowing) has attracted considerable attention, because it was once thought to be a rare cause of hypertension, but is now known to be the most common cause of secondary hypertension. In The Kidney and Hypertension, George Bakris and a host of international contributors examine all aspects of hypertension in the renal patient.
A neurogenic component to primary hypertension (hypertension) is now well established. Cabassi A, Vinci S, Calzolari M, Bruschi G, Borghetti A. Regional sympathetic activity in pre-hypertensive phase of spontaneously hypertensive rats. Life Sci 1998; 62 (12): 1111–1118. Along with raised vasomotor tone and increased cardiac output, the chronic activation of the sympathetic nervous system in hypertension has a diverse range of pathophysiological consequences independent of any increase in blood pressure.